Submit Data |  Help |  Video Tutorials |  News |  Publications |  FTP Download |  REST API |  Citing RGD |  Contact   

ONTOLOGY REPORT - ANNOTATIONS


Term:negative regulation of type II hypersensitivity
go back to main search page
Accession:GO:0002893 term browser browse the term
Definition:Any process that stops, prevents, or reduces the frequency, rate, or extent of type II hypersensitivity.
Synonyms:exact_synonym: down regulation of type II hypersensitivity;   downregulation of type II hypersensitivity
 narrow_synonym: inhibition of type II hypersensitivity


show annotations for term's descendants       view all columns           Sort by:
 

Term paths to the root
Path 1
Term Annotations click to browse term
  biological_process 19578
    immune system process 2693
      immune effector process 861
        leukocyte mediated immunity 502
          myeloid leukocyte mediated immunity 117
            type II hypersensitivity 6
              negative regulation of type II hypersensitivity 0
                negative regulation of type IIa hypersensitivity + 0
                negative regulation of type IIb hypersensitivity 0
Path 2
Term Annotations click to browse term
  biological_process 19578
    biological regulation 13539
      regulation of biological process 12843
        regulation of response to stimulus 4027
          regulation of response to stress 1366
            regulation of defense response 619
              regulation of inflammatory response 370
                regulation of acute inflammatory response 55
                  negative regulation of acute inflammatory response 13
                    negative regulation of acute inflammatory response to antigenic stimulus 7
                      negative regulation of hypersensitivity 3
                        negative regulation of type II hypersensitivity 0
                          negative regulation of type IIa hypersensitivity + 0
                          negative regulation of type IIb hypersensitivity 0
paths to the root

NHLBI Logo

RGD is funded by grant HL64541 from the National Heart, Lung, and Blood Institute on behalf of the NIH.