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ONTOLOGY REPORT - ANNOTATIONS


Term:regulation of collateral sprouting of intact axon in response to injury
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Accession:GO:0048683 term browser browse the term
Definition:Any process that modulates the frequency, rate or extent of collateral sprouting of an intact axon as a result of injury to an axon.


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negative regulation of collateral sprouting of intact axon in response to injury term browser
Symbol Object Name JBrowse Chr Start Stop Reference
G Spp1 secreted phosphoprotein 1 JBrowse link NW_004936905 148,956 156,023 RGD:9068941

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Path 1
Term Annotations click to browse term
  biological_process 11930
    growth 972
      regulation of growth 626
        regulation of developmental growth 371
          regulation of collateral sprouting 23
            regulation of collateral sprouting of intact axon in response to injury 1
              negative regulation of collateral sprouting of intact axon in response to injury 1
              positive regulation of collateral sprouting of intact axon in response to injury 0
Path 2
Term Annotations click to browse term
  biological_process 11930
    developmental process 5576
      anatomical structure development 5218
        multicellular organism development 4786
          system development 4532
            nervous system development 2348
              neurogenesis 1688
                generation of neurons 1565
                  neuron differentiation 1405
                    neuron development 1150
                      neuron projection development 1020
                        neuron projection morphogenesis 605
                          axonogenesis 396
                            collateral sprouting 31
                              regulation of collateral sprouting 23
                                regulation of collateral sprouting of intact axon in response to injury 1
                                  negative regulation of collateral sprouting of intact axon in response to injury 1
                                  positive regulation of collateral sprouting of intact axon in response to injury 0
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RGD is funded by grant HL64541 from the National Heart, Lung, and Blood Institute on behalf of the NIH.