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ONTOLOGY REPORT - ANNOTATIONS


Term:negative regulation of glucocorticoid mediated signaling pathway
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Accession:GO:1900170 term browser browse the term
Definition:Any process that stops, prevents or reduces the frequency, rate or extent of glucocorticoid mediated signaling pathway.
Synonyms:exact_synonym: down regulation of glucocorticoid mediated signaling pathway;   down regulation of glucocorticoid mediated signalling;   downregulation of glucocorticoid mediated signaling pathway;   downregulation of glucocorticoid mediated signalling;   negative regulation of glucocorticoid mediated signalling
 narrow_synonym: inhibition of glucocorticoid mediated signaling pathway;   inhibition of glucocorticoid mediated signalling


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negative regulation of glucocorticoid mediated signaling pathway term browser
Symbol Object Name JBrowse Chr Start Stop Reference
G Crebrf CREB3 regulatory factor JBrowse link 10 16,693,846 16,753,162 RGD:1624291
G Nr3c1 nuclear receptor subfamily 3, group C, member 1 JBrowse link 18 31,728,373 32,704,022 RGD:1624291

Term paths to the root
Path 1
Term Annotations click to browse term
  biological_process 19858
    cellular process 18618
      signal transduction 6968
        negative regulation of signal transduction 1232
          negative regulation of glucocorticoid mediated signaling pathway 2
Path 2
Term Annotations click to browse term
  biological_process 19858
    response to stimulus 10526
      response to chemical 6305
        response to organic substance 3832
          response to hormone 1206
            response to steroid hormone 514
              response to corticosteroid 327
                response to glucocorticoid 302
                  cellular response to glucocorticoid stimulus 123
                    glucocorticoid mediated signaling pathway 5
                      regulation of glucocorticoid mediated signaling pathway 3
                        negative regulation of glucocorticoid mediated signaling pathway 2
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RGD is funded by grant HL64541 from the National Heart, Lung, and Blood Institute on behalf of the NIH.