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Ontology Browser

Term:
regulation of intrinsic apoptotic signaling pathway by p53 class mediator (GO:1902253)
Annotations: Rat: (32) Mouse: (32) Human: (36) Chinchilla: (31) Bonobo: (32) Dog: (31) Squirrel: (31) Pig: (32)
Parent Terms Term With Siblings Child Terms
intrinsic apoptotic signaling pathway in response to DNA damage by p53 class mediator +   
intrinsic apoptotic signaling pathway in response to osmotic stress by p53 class mediator +   
negative regulation of intrinsic apoptotic signaling pathway +   
negative regulation of intrinsic apoptotic signaling pathway by p53 class mediator +   
negative regulation of signal transduction by p53 class mediator +   
positive regulation of intrinsic apoptotic signaling pathway +   
positive regulation of intrinsic apoptotic signaling pathway by p53 class mediator +   
positive regulation of signal transduction by p53 class mediator +   
regulation of DNA damage response, signal transduction by p53 class mediator +   
regulation of endoplasmic reticulum stress-induced intrinsic apoptotic signaling pathway +   
regulation of hypoxia-induced intrinsic apoptotic signaling pathway +   
regulation of intrinsic apoptotic signaling pathway by p53 class mediator +   
Any process that modulates the frequency, rate or extent of intrinsic apoptotic signaling pathway by p53 class mediator.
regulation of intrinsic apoptotic signaling pathway in response to DNA damage +   
regulation of intrinsic apoptotic signaling pathway in response to osmotic stress +   
regulation of nitrosative stress-induced intrinsic apoptotic signaling pathway +   
regulation of oxidative stress-induced intrinsic apoptotic signaling pathway +   

Synonyms
Exact Synonyms: regulation of intrinsic apoptotic signaling pathway by signal transduction by p53 class mediator
Related Synonyms: regulation of signal transduction by p53 class mediator resulting in induction of apoptosis
Definition Sources: GOC:BHF, GOC:mtg_apoptosis, GOC:rl, GOC:TermGenie, PMID:15705871

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RGD is funded by grant HL64541 from the National Heart, Lung, and Blood Institute on behalf of the NIH.