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GENE - TERM ANNOTATION REPORT

3 Annotations Found.

An association has been curated linking Rnf152 and negative regulation of TORC1 signaling in Rattus norvegicus.        

  • The association was inferred from sequence orthology (ISO)
  •  
  • The annotation was made from RGD pipelines: Rat ISO annotations derived from the GO annotations of orthologous genes in other species.
  • The annotation has been inferred from sequence orthology with RNF152 (Homo sapiens)
  • 18 CRRD objects have been annotated to negative regulation of TORC1 signaling  (GO:1904262)
  • 2 papers in CRRD have been used to annotate Rnf152
  • Curation Notes: (PMID:25936802)
  • Original References(s): PMID:25936802


  • An association has been curated linking Rnf152 and negative regulation of TORC1 signaling in Rattus norvegicus.        

  • The association was inferred from sequence orthology (ISO)
  •  
  • The annotation was made from RGD pipelines: Rat ISO annotations derived from the GO annotations of orthologous genes in other species.
  • The annotation has been inferred from sequence orthology with Rnf152 (Mus musculus)
  • 18 CRRD objects have been annotated to negative regulation of TORC1 signaling  (GO:1904262)
  • 2 papers in CRRD have been used to annotate Rnf152
  • Curation Notes: (MGI:MGI:5644195|PMID:25936802)
  • Original References(s): MGI:MGI:5644195, PMID:25936802


  • An association has been curated linking Rnf152 and negative regulation of TORC1 signaling in Rattus norvegicus.        

  • The association was inferred from sequence or structural similarity (ISS)
  •  
  • The annotation was made from RGD automated data pipeline
  • The annotation has been inferred from sequence or structural similarity with UniProtKB:Q8N8N0
  • 18 CRRD objects have been annotated to negative regulation of TORC1 signaling  (GO:1904262)
  • 2 papers in CRRD have been used to annotate Rnf152
  • Curation Notes: GO_REF:0000024
  • Original References(s): GO_REF:0000024


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    RGD is funded by grant HL64541 from the National Heart, Lung, and Blood Institute on behalf of the NIH.