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Gene: Ralgapa2 (Ral GTPase activating protein catalytic alpha subunit 2) Rattus norvegicus
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Symbol: Ralgapa2
Name: Ral GTPase activating protein catalytic alpha subunit 2
Description: Exhibits GTPase activator activity and protein heterodimerization activity. Involved in activation of GTPase activity. Predicted to localize to the cytosol and plasma membrane. Orthologous to human RALGAPA2 (Ral GTPase activating protein catalytic alpha subunit 2); INTERACTS WITH 2,5-hexanedione; bisphenol A; Monobutylphthalate.
Type: protein-coding
RefSeq Status: MODEL
Also known as: 250 kDa substrate of Akt; AS250; LOC296211; p220; Ral GTPase activating protein, alpha subunit 2 (catalytic); Ral GTPase-activating protein alpha subunit 2; ral GTPase-activating protein subunit alpha-2; RGD1308023; similar to CG5521-PA
Orthologs:
Latest Assembly: Rnor_6.0 - RGSC Genome Assembly v6.0
Position:
Rat AssemblyChrPosition (strand)SourceGenome Browsers
JBrowseNCBIUCSCEnsembl
Rnor_6.03140,451,850 - 140,728,305 (-)NCBIRnor6.0Rnor_6.0rn6Rnor6.0
Rnor_5.03146,870,901 - 147,146,723 (-)NCBIRnor5.0Rnor_5.0rn5Rnor5.0
RGSC_v3.43134,867,846 - 135,145,583 (-)NCBIRGSC3.4rn4RGSC3.4
RGSC_v3.13134,893,509 - 134,996,492 (-)NCBI
Celera3132,526,330 - 132,803,310 (-)NCBICelera
Cytogenetic Map3q41NCBI
JBrowse: View Region in Genome Browser (JBrowse)
Model


Gene-Chemical Interaction Annotations
Gene Ontology Annotations
References - curated
References - uncurated

Genomics

Comparative Map Data
Position Markers
QTLs in Region (Rnor_6.0)
miRNA Target Status

Sequence

Nucleotide Sequences
Protein Sequences
Transcriptome

Strain Variation

Strain Sequence Variants (Rnor 5.0)
Damaging Variants

Additional Information

External Database Links
Nomenclature History
 
More on Ralgapa2
Alliance Gene
NCBI Gene
Ensembl Gene
JBrowse: rn5 rn6
NCBI Genome Data Viewer

CRRD Object Information
CRRD ID: 1308023
Created: 2005-01-12
Species: Rattus norvegicus
Last Modified: 2019-04-23
Status: ACTIVE



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RGD is funded by grant HL64541 from the National Heart, Lung, and Blood Institute on behalf of the NIH.