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Gene: Ctif (cap binding complex dependent translation initiation factor) Rattus norvegicus
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Symbol: Ctif
Name: cap binding complex dependent translation initiation factor
Description: Predicted to have translation activator activity. Predicted to be involved in nuclear-transcribed mRNA catabolic process, nonsense-mediated decay and regulation of translational initiation. Predicted to localize to the cytosol and perinuclear region of cytoplasm. Orthologous to human CTIF (cap binding complex dependent translation initiation factor); INTERACTS WITH bisphenol A; thioacetamide; (-)-epigallocatechin 3-gallate (ortholog).
Type: protein-coding
RefSeq Status: PROVISIONAL
Also known as: CBP80/20-dependent translation initiation factor; gene model 672; gene model 672, (NCBI); Gm672; LOC364900
Orthologs:
Latest Assembly: Rnor_6.0 - RGSC Genome Assembly v6.0
Position:
Rat AssemblyChrPosition (strand)SourceGenome Browsers
JBrowseNCBIUCSCEnsembl
Rnor_6.01871,467,381 - 71,701,423 (-)NCBIRnor6.0Rnor_6.0rn6Rnor6.0
Rnor_5.01870,602,149 - 70,834,596 (-)NCBIRnor5.0Rnor_5.0rn5Rnor5.0
RGSC_v3.41872,366,505 - 72,458,414 (-)NCBIRGSC3.4rn4RGSC3.4
RGSC_v3.11872,439,430 - 72,657,616 (-)NCBI
Celera1867,226,619 - 67,314,620 (-)NCBICelera
Cytogenetic Map18q12.2NCBI
JBrowse: View Region in Genome Browser (JBrowse)
Model


Gene-Chemical Interaction Annotations
Gene Ontology Annotations
References - curated
References - uncurated

Genomics

Comparative Map Data
Position Markers
QTLs in Region (Rnor_6.0)
miRNA Target Status

Sequence

Nucleotide Sequences
Protein Sequences
Transcriptome

Strain Variation

Strain Sequence Variants (Rnor 6.0)
Damaging Variants

Additional Information

External Database Links
Nomenclature History
 
More on Ctif
Alliance Gene
NCBI Gene
Ensembl Gene
JBrowse: rn5 rn6
NCBI Genome Data Viewer

CRRD Object Information
CRRD ID: 1311887
Created: 2005-01-12
Species: Rattus norvegicus
Last Modified: 2019-08-20
Status: ACTIVE



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RGD is funded by grant HL64541 from the National Heart, Lung, and Blood Institute on behalf of the NIH.