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Gene: Hist1h2ak (histone cluster 1, H2ak) Rattus norvegicus
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Symbol: Hist1h2ak
Name: histone cluster 1, H2ak
Description: Predicted to have DNA binding activity. Predicted to be involved in chromatin silencing and negative regulation of cell population proliferation. Predicted to localize to nuclear chromatin. Orthologous to human H2AC4 (H2A clustered histone 4); PARTICIPATES IN systemic lupus erythematosus pathway; INTERACTS WITH 6-propyl-2-thiouracil; bisphenol A; phenformin.
Type: protein-coding
RefSeq Status: PROVISIONAL
Also known as: LOC680615; similar to histone 2a
Orthologs:
Homo sapiens (human) : H2AC4 (H2A clustered histone 4)  HGNC  Alliance
Mus musculus (house mouse) : H2ac4 (H2A clustered histone 4)  MGI  Alliance
more info ...
Latest Assembly: Rnor_6.0 - RGSC Genome Assembly v6.0
Position:
Rat AssemblyChrPosition (strand)SourceGenome Browsers
JBrowseNCBIUCSCEnsembl
Rnor_6.01744,840,990 - 44,841,382 (-)NCBIRnor6.0Rnor_6.0rn6Rnor6.0
Rnor_6.0 Ensembl1744,840,990 - 44,841,382 (-)EnsemblRnor6.0rn6Rnor6.0
Rnor_5.01758,624,879 - 58,625,271 (+)NCBIRnor5.0Rnor_5.0rn5Rnor5.0
RGSC_v3.41750,436,332 - 50,436,724 (-)NCBIRGSC3.4rn4RGSC3.4
Celera1753,661,266 - 53,661,658 (+)NCBICelera
Cytogenetic Map17p11NCBI
JBrowse: View Region in Genome Browser (JBrowse)
Model


Gene-Chemical Interaction Annotations
Gene Ontology Annotations
Molecular Pathway Annotations
References - curated
References - uncurated

Genomics

Comparative Map Data
QTLs in Region (Rnor_6.0)
miRNA Target Status

Expression

RNA-SEQ Expression

Sequence

Nucleotide Sequences
Protein Sequences
Transcriptome
Promoters

Strain Variation

Strain Sequence Variants (Rnor 6.0)
Damaging Variants

Additional Information

External Database Links
Nomenclature History
 
More on Hist1h2ak
Alliance Gene
NCBI Gene
Ensembl Gene
JBrowse: rn5 rn6
NCBI Genome Data Viewer

CRRD Object Information
CRRD ID: 1589461
Created: 2006-11-19
Species: Rattus norvegicus
Last Modified: 2020-06-09
Status: ACTIVE



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RGD is funded by grant HL64541 from the National Heart, Lung, and Blood Institute on behalf of the NIH.